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doi: 10.1021/jf071303g 30

Much evidence suggests that activation of anorexogenic proopiomelanocortin (POMC) neurons and simultaneous inhibition of orexigenic neuropeptide Y/Agouti-related peptide (NPY/AgRP) neurons in the ARC appears to be one of the major mechanisms for GLP-1R mediated inhibition of food intake

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GHK-Cu: not FDA-approved as a drug What Each Peptide Does Independently Before evaluating whether these two agents make sense together, it is worth understanding what each one does on its own, how the evidence grades out, and where the mechanistic story is solid versus speculative